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Corrigendum to "Inhibition of AKT sensitizes chemoresistant ovarian cancer cells to cisplatin by abrogating S and G2/M arrest" (Exp. Mol. Pathol. 2016 100(3) 506-13).

Experimental and molecular pathology

Zhang D, Piao HL, Li YH, Qiu Q, Li DJ, Du MR, Tsang BK.
PMID: 27392408
Exp Mol Pathol. 2016 Aug;101(1):163. doi: 10.1016/j.yexmp.2016.06.004. Epub 2016 Jul 06.

No abstract available.

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Zhang D, Piao HL, Li YH, et al. Corrigendum to "Inhibition of AKT sensitizes chemoresistant ovarian cancer cells to cisplatin by abrogating S and G2/M arrest" (Exp. Mol. Pathol. 2016 100(3) 506-13). Exp Mol Pathol. 2016;101(1):163doi: 10.1016/j.yexmp.2016.06.004.
Zhang, D., Piao, H. L., Li, Y. H., Qiu, Q., Li, D. J., Du, M. R., & Tsang, B. K. (2016). Corrigendum to "Inhibition of AKT sensitizes chemoresistant ovarian cancer cells to cisplatin by abrogating S and G2/M arrest" (Exp. Mol. Pathol. 2016 100(3) 506-13). Experimental and molecular pathology, 101(1), 163. https://doi.org/10.1016/j.yexmp.2016.06.004
Zhang, Di, et al. "Corrigendum to "Inhibition of AKT sensitizes chemoresistant ovarian cancer cells to cisplatin by abrogating S and G2/M arrest" (Exp. Mol. Pathol. 2016 100(3) 506-13)." Experimental and molecular pathology vol. 101,1 (2016): 163. doi: https://doi.org/10.1016/j.yexmp.2016.06.004
Zhang D, Piao HL, Li YH, Qiu Q, Li DJ, Du MR, Tsang BK. Corrigendum to "Inhibition of AKT sensitizes chemoresistant ovarian cancer cells to cisplatin by abrogating S and G2/M arrest" (Exp. Mol. Pathol. 2016 100(3) 506-13). Exp Mol Pathol. 2016 Aug;101(1):163. doi: 10.1016/j.yexmp.2016.06.004. Epub 2016 Jul 06. PMID: 27392408.
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Corrigendum to "AQP4 and HIVAN" Experimental and Molecular Pathology, 105(2018) 71-75.

Experimental and molecular pathology

Mubariz F, Bryant JL, Nimmagadda VKC, Ray J, Makar TK.
PMID: 30360924
Exp Mol Pathol. 2018 Dec;105(3):417. doi: 10.1016/j.yexmp.2018.08.007. Epub 2018 Oct 22.

No abstract available.

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Mubariz F, Bryant JL, Nimmagadda VKC, et al. Corrigendum to "AQP4 and HIVAN" Experimental and Molecular Pathology, 105(2018) 71-75. Exp Mol Pathol. 2018;105(3):417doi: 10.1016/j.yexmp.2018.08.007.
Mubariz, F., Bryant, J. L., Nimmagadda, V. K. C., Ray, J., & Makar, T. K. (2018). Corrigendum to "AQP4 and HIVAN" Experimental and Molecular Pathology, 105(2018) 71-75. Experimental and molecular pathology, 105(3), 417. https://doi.org/10.1016/j.yexmp.2018.08.007
Mubariz, Fahad, et al. "Corrigendum to "AQP4 and HIVAN" Experimental and Molecular Pathology, 105(2018) 71-75." Experimental and molecular pathology vol. 105,3 (2018): 417. doi: https://doi.org/10.1016/j.yexmp.2018.08.007
Mubariz F, Bryant JL, Nimmagadda VKC, Ray J, Makar TK. Corrigendum to "AQP4 and HIVAN" Experimental and Molecular Pathology, 105(2018) 71-75. Exp Mol Pathol. 2018 Dec;105(3):417. doi: 10.1016/j.yexmp.2018.08.007. Epub 2018 Oct 22. PMID: 30360924.
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Retracted: Resveratrol inhibits oesophageal adenocarcinoma cell proliferation via AMP-activated protein kinase signaling.

Asian Pacific journal of cancer prevention : APJCP

[No authors listed]
PMID: 29171745
Asian Pac J Cancer Prev. 2017 Nov 24;18(10):2895-2895. doi: 10.22034/apjcp.2017.18.10.2895.

Retraction: Retracted:Resveratrol inhibits oesophageal adenocarcinoma cell proliferation via AMP-activated protein kinase signaling Asian Pacific Journal of Cancer Prevention (APJCP) has retracted the article titled “Resveratrol Inhibits Oesophageal Adenocarcinoma Cell Proliferation via AMP-activated Protein Kinase Signaling”(1) for reason of having...

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Retracted: Resveratrol inhibits oesophageal adenocarcinoma cell proliferation via AMP-activated protein kinase signaling. Asian Pac J Cancer Prev. 2017;18(10):2895-2895doi: 10.22034/apjcp.2017.18.10.2895.
(2017). Retracted: Resveratrol inhibits oesophageal adenocarcinoma cell proliferation via AMP-activated protein kinase signaling. Asian Pacific journal of cancer prevention : APJCP, 18(10), 2895-2895. https://doi.org/10.22034/apjcp.2017.18.10.2895
"Retracted: Resveratrol inhibits oesophageal adenocarcinoma cell proliferation via AMP-activated protein kinase signaling." Asian Pacific journal of cancer prevention : APJCP vol. 18,10 (2017): 2895-2895. doi: https://doi.org/10.22034/apjcp.2017.18.10.2895
Retracted: Resveratrol inhibits oesophageal adenocarcinoma cell proliferation via AMP-activated protein kinase signaling. Asian Pac J Cancer Prev. 2017 Nov 24;18(10):2895-2895. doi: 10.22034/apjcp.2017.18.10.2895. PMID: 29171745; PMCID: PMC5747421.
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WITHDRAWN: Gold coated dual-resonance long-period fiber gratings (DR-LPFG) based Aptasensor for Cyanobacterial toxin detection.

Experimental and molecular pathology

Tripathi SM, Dandapat K, Bock WJ, Mikulic P, Perreault J, Sellamuthu B.
PMID: 31145887
Exp Mol Pathol. 2019 May 28;104266. doi: 10.1016/j.yexmp.2019.104266. Epub 2019 May 28.

No abstract available.

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Tripathi SM, Dandapat K, Bock WJ, et al. WITHDRAWN: Gold coated dual-resonance long-period fiber gratings (DR-LPFG) based Aptasensor for Cyanobacterial toxin detection. Exp Mol Pathol. 2019;104266doi: 10.1016/j.yexmp.2019.104266.
Tripathi, S. M., Dandapat, K., Bock, W. J., Mikulic, P., Perreault, J., & Sellamuthu, B. (2019). WITHDRAWN: Gold coated dual-resonance long-period fiber gratings (DR-LPFG) based Aptasensor for Cyanobacterial toxin detection. Experimental and molecular pathology, 104266. https://doi.org/10.1016/j.yexmp.2019.104266
Tripathi, Saurabh Mani, et al. "WITHDRAWN: Gold coated dual-resonance long-period fiber gratings (DR-LPFG) based Aptasensor for Cyanobacterial toxin detection." Experimental and molecular pathology vol. (2019): 104266. doi: https://doi.org/10.1016/j.yexmp.2019.104266
Tripathi SM, Dandapat K, Bock WJ, Mikulic P, Perreault J, Sellamuthu B. WITHDRAWN: Gold coated dual-resonance long-period fiber gratings (DR-LPFG) based Aptasensor for Cyanobacterial toxin detection. Exp Mol Pathol. 2019 May 28;104266. doi: 10.1016/j.yexmp.2019.104266. Epub 2019 May 28. PMID: 31145887.
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Critical roles of microRNA-196 in normal physiology and non-malignant diseases: Diagnostic and therapeutic implications.

Experimental and molecular pathology

Bastami M, Masotti A, Saadatian Z, Daraei A, Farjam M, Ghanbariasad A, Vahed SZ, Eyvazi S, Mansoori Y, Nariman-Saleh-Fam Z.
PMID: 34166682
Exp Mol Pathol. 2021 Oct;122:104664. doi: 10.1016/j.yexmp.2021.104664. Epub 2021 Jun 21.

MicroRNAs (miRNAs) have emerged as a critical component of regulatory networks that modulate and fine-tune gene expression in a post-transcriptional manner. The microRNA-196 family is encoded by three loci in the human genome, namely hsa-mir-196a-1, hsa-mir-196a-2, and hsa-mir-196b. Increasing...

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Bastami M, Masotti A, Saadatian Z, et al. Critical roles of microRNA-196 in normal physiology and non-malignant diseases: Diagnostic and therapeutic implications. Exp Mol Pathol. 2021;122:104664doi: 10.1016/j.yexmp.2021.104664.
Bastami, M., Masotti, A., Saadatian, Z., Daraei, A., Farjam, M., Ghanbariasad, A., Vahed, S. Z., Eyvazi, S., Mansoori, Y., & Nariman-Saleh-Fam, Z. (2021). Critical roles of microRNA-196 in normal physiology and non-malignant diseases: Diagnostic and therapeutic implications. Experimental and molecular pathology, 122104664. https://doi.org/10.1016/j.yexmp.2021.104664
Bastami, Milad, et al. "Critical roles of microRNA-196 in normal physiology and non-malignant diseases: Diagnostic and therapeutic implications." Experimental and molecular pathology vol. 122 (2021): 104664. doi: https://doi.org/10.1016/j.yexmp.2021.104664
Bastami M, Masotti A, Saadatian Z, Daraei A, Farjam M, Ghanbariasad A, Vahed SZ, Eyvazi S, Mansoori Y, Nariman-Saleh-Fam Z. Critical roles of microRNA-196 in normal physiology and non-malignant diseases: Diagnostic and therapeutic implications. Exp Mol Pathol. 2021 Oct;122:104664. doi: 10.1016/j.yexmp.2021.104664. Epub 2021 Jun 21. PMID: 34166682.
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Gene expression with corresponding pathways analysis in Gaucher disease.

Experimental and molecular pathology

Pawliński Ł, Polus A, Kałużna M, Sordyl M, Tobór-Świętek E, Krawczyk M, Bednarek M, Solnica B, Ruchała M, Kieć-Wilk B.
PMID: 34481839
Exp Mol Pathol. 2021 Dec;123:104679. doi: 10.1016/j.yexmp.2021.104679. Epub 2021 Sep 02.

Gaucher disease (GD) caused by mutation in the GBA gene has a wide spectrum of phenotypes. Besides the storage disorder, secondary alteration of various pathways occurs with modification of the expression of many genes. In our work we analysed...

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Pawliński Ł, Polus A, Kałużna M, et al. Gene expression with corresponding pathways analysis in Gaucher disease. Exp Mol Pathol. 2021;123:104679doi: 10.1016/j.yexmp.2021.104679.
Pawliński, �. �., Polus, A., Kałużna, M., Sordyl, M., Tobór-Świętek, E., Krawczyk, M., Bednarek, M., Solnica, B., Ruchała, M., & Kieć-Wilk, B. (2021). Gene expression with corresponding pathways analysis in Gaucher disease. Experimental and molecular pathology, 123104679. https://doi.org/10.1016/j.yexmp.2021.104679
Pawliński, Łukasz, et al. "Gene expression with corresponding pathways analysis in Gaucher disease." Experimental and molecular pathology vol. 123 (2021): 104679. doi: https://doi.org/10.1016/j.yexmp.2021.104679
Pawliński Ł, Polus A, Kałużna M, Sordyl M, Tobór-Świętek E, Krawczyk M, Bednarek M, Solnica B, Ruchała M, Kieć-Wilk B. Gene expression with corresponding pathways analysis in Gaucher disease. Exp Mol Pathol. 2021 Dec;123:104679. doi: 10.1016/j.yexmp.2021.104679. Epub 2021 Sep 02. PMID: 34481839.
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HSPB8 promoted intrahepatic cholangiocarcinoma progression by enhancing epithelial-mesenchymal transition and autophagy.

Experimental and molecular pathology

Shu B, Zhou Y, Liang Q, He C, Li F.
PMID: 34606781
Exp Mol Pathol. 2021 Dec;123:104691. doi: 10.1016/j.yexmp.2021.104691. Epub 2021 Oct 01.

PURPOSE: Heat shock protein B8 (HSPB8) has been recently discovered to be participated in the regulation of tumor progression. However, the function of HSPB8 in intrahepatic cholangiocarcinoma (ICC) has not yet been elucidated. This study studied the function of...

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Shu B, Zhou Y, Liang Q, et al. HSPB8 promoted intrahepatic cholangiocarcinoma progression by enhancing epithelial-mesenchymal transition and autophagy. Exp Mol Pathol. 2021;123:104691doi: 10.1016/j.yexmp.2021.104691.
Shu, B., Zhou, Y., Liang, Q., He, C., & Li, F. (2021). HSPB8 promoted intrahepatic cholangiocarcinoma progression by enhancing epithelial-mesenchymal transition and autophagy. Experimental and molecular pathology, 123104691. https://doi.org/10.1016/j.yexmp.2021.104691
Shu, Bo, et al. "HSPB8 promoted intrahepatic cholangiocarcinoma progression by enhancing epithelial-mesenchymal transition and autophagy." Experimental and molecular pathology vol. 123 (2021): 104691. doi: https://doi.org/10.1016/j.yexmp.2021.104691
Shu B, Zhou Y, Liang Q, He C, Li F. HSPB8 promoted intrahepatic cholangiocarcinoma progression by enhancing epithelial-mesenchymal transition and autophagy. Exp Mol Pathol. 2021 Dec;123:104691. doi: 10.1016/j.yexmp.2021.104691. Epub 2021 Oct 01. PMID: 34606781.
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The combination of TRAIL and the Smac mimetic LCL-161 induces an irreversible phenotypic change of MCF-7 breast cancer cells.

Experimental and molecular pathology

Granqvist V, Holmgren C, Larsson C.
PMID: 35007560
Exp Mol Pathol. 2022 Jan 07;104739. doi: 10.1016/j.yexmp.2021.104739. Epub 2022 Jan 07.

BACKGROUND: Breast cancer is the most common malignancy affecting women. Although the prognosis generally is good, a substantial number of patients still suffer from relapse, emphasizing the need for novel treatments. Smac mimetics were developed to facilitate cell death...

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Granqvist V, Holmgren C, Larsson C. The combination of TRAIL and the Smac mimetic LCL-161 induces an irreversible phenotypic change of MCF-7 breast cancer cells. Exp Mol Pathol. 2022;104739doi: 10.1016/j.yexmp.2021.104739.
Granqvist, V., Holmgren, C., & Larsson, C. (2022). The combination of TRAIL and the Smac mimetic LCL-161 induces an irreversible phenotypic change of MCF-7 breast cancer cells. Experimental and molecular pathology, 104739. https://doi.org/10.1016/j.yexmp.2021.104739
Granqvist, Victoria, et al. "The combination of TRAIL and the Smac mimetic LCL-161 induces an irreversible phenotypic change of MCF-7 breast cancer cells." Experimental and molecular pathology vol. (2022): 104739. doi: https://doi.org/10.1016/j.yexmp.2021.104739
Granqvist V, Holmgren C, Larsson C. The combination of TRAIL and the Smac mimetic LCL-161 induces an irreversible phenotypic change of MCF-7 breast cancer cells. Exp Mol Pathol. 2022 Jan 07;104739. doi: 10.1016/j.yexmp.2021.104739. Epub 2022 Jan 07. PMID: 35007560.
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Setting a diagnostic benchmark for tumor BRCA testing: detection of BRCA1 and BRCA2 large genomic rearrangements in FFPE tissue - A pilot study.

Experimental and molecular pathology

Valtcheva N, Nguyen-Sträuli BD, Wagner U, Freiberger SN, Varga Z, Britschgi C, Dedes KJ, Rechsteiner MP.
PMID: 34637782
Exp Mol Pathol. 2021 Dec;123:104705. doi: 10.1016/j.yexmp.2021.104705. Epub 2021 Oct 09.

PARP inhibitors are used for treatment of tumors lacking function of the double-strand DNA break repair proteins BRCA1 or BRCA2 and are already approved for several cancer types. Thus, it is clinically crucial to determine germline as well as...

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Valtcheva N, Nguyen-Sträuli BD, Wagner U, et al. Setting a diagnostic benchmark for tumor BRCA testing: detection of BRCA1 and BRCA2 large genomic rearrangements in FFPE tissue - A pilot study. Exp Mol Pathol. 2021;123:104705doi: 10.1016/j.yexmp.2021.104705.
Valtcheva, N., Nguyen-Sträuli, B. D., Wagner, U., Freiberger, S. N., Varga, Z., Britschgi, C., Dedes, K. J., & Rechsteiner, M. P. (2021). Setting a diagnostic benchmark for tumor BRCA testing: detection of BRCA1 and BRCA2 large genomic rearrangements in FFPE tissue - A pilot study. Experimental and molecular pathology, 123104705. https://doi.org/10.1016/j.yexmp.2021.104705
Valtcheva, Nadejda, et al. "Setting a diagnostic benchmark for tumor BRCA testing: detection of BRCA1 and BRCA2 large genomic rearrangements in FFPE tissue - A pilot study." Experimental and molecular pathology vol. 123 (2021): 104705. doi: https://doi.org/10.1016/j.yexmp.2021.104705
Valtcheva N, Nguyen-Sträuli BD, Wagner U, Freiberger SN, Varga Z, Britschgi C, Dedes KJ, Rechsteiner MP. Setting a diagnostic benchmark for tumor BRCA testing: detection of BRCA1 and BRCA2 large genomic rearrangements in FFPE tissue - A pilot study. Exp Mol Pathol. 2021 Dec;123:104705. doi: 10.1016/j.yexmp.2021.104705. Epub 2021 Oct 09. PMID: 34637782.
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The role of adipokines in leiomyomas development.

Experimental and molecular pathology

Strzałkowska B, Dawidowicz M, Ochman B, Świętochowska E.
PMID: 34637783
Exp Mol Pathol. 2021 Dec;123:104693. doi: 10.1016/j.yexmp.2021.104693. Epub 2021 Oct 09.

INTRODUCTION: Many women of premenopausal age suffer from uterine leiomyomas, which are benign tumors of the uterus. Despite the high prevalence of uterine leiomyomas underlying pathogenesis mechanisms are not fully elucidated. Early data showed a positive correlation between increased...

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Strzałkowska B, Dawidowicz M, Ochman B, et al. The role of adipokines in leiomyomas development. Exp Mol Pathol. 2021;123:104693doi: 10.1016/j.yexmp.2021.104693.
Strzałkowska, B., Dawidowicz, M., Ochman, B., & Świętochowska, E. (2021). The role of adipokines in leiomyomas development. Experimental and molecular pathology, 123104693. https://doi.org/10.1016/j.yexmp.2021.104693
Strzałkowska, Bogumiła, et al. "The role of adipokines in leiomyomas development." Experimental and molecular pathology vol. 123 (2021): 104693. doi: https://doi.org/10.1016/j.yexmp.2021.104693
Strzałkowska B, Dawidowicz M, Ochman B, Świętochowska E. The role of adipokines in leiomyomas development. Exp Mol Pathol. 2021 Dec;123:104693. doi: 10.1016/j.yexmp.2021.104693. Epub 2021 Oct 09. PMID: 34637783.
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The role of cell surface proteins gene expression in diagnosis, prognosis, and drug resistance of colorectal cancer: In silico analysis and validation.

Experimental and molecular pathology

Nazempour N, Taleqani MH, Taheri N, Haji Ali Asgary Najafabadi AH, Shokrollahi A, Zamani A, Fattahi Dolatabadi N, Peymani M, Mahdevar M.
PMID: 34592197
Exp Mol Pathol. 2021 Dec;123:104688. doi: 10.1016/j.yexmp.2021.104688. Epub 2021 Sep 27.

Cell surface proteins (CSPs) are an important type of protein in different essential cell functions. This study aimed to distinguish overexpressed CSPs in colorectal cancer to investigate their biomarker, prognosis, and drug resistance potential. Raw data of three datasets...

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Nazempour N, Taleqani MH, Taheri N, et al. The role of cell surface proteins gene expression in diagnosis, prognosis, and drug resistance of colorectal cancer: In silico analysis and validation. Exp Mol Pathol. 2021;123:104688doi: 10.1016/j.yexmp.2021.104688.
Nazempour, N., Taleqani, M. H., Taheri, N., Haji Ali Asgary Najafabadi, A. H., Shokrollahi, A., Zamani, A., Fattahi Dolatabadi, N., Peymani, M., & Mahdevar, M. (2021). The role of cell surface proteins gene expression in diagnosis, prognosis, and drug resistance of colorectal cancer: In silico analysis and validation. Experimental and molecular pathology, 123104688. https://doi.org/10.1016/j.yexmp.2021.104688
Nazempour, Nasrin, et al. "The role of cell surface proteins gene expression in diagnosis, prognosis, and drug resistance of colorectal cancer: In silico analysis and validation." Experimental and molecular pathology vol. 123 (2021): 104688. doi: https://doi.org/10.1016/j.yexmp.2021.104688
Nazempour N, Taleqani MH, Taheri N, Haji Ali Asgary Najafabadi AH, Shokrollahi A, Zamani A, Fattahi Dolatabadi N, Peymani M, Mahdevar M. The role of cell surface proteins gene expression in diagnosis, prognosis, and drug resistance of colorectal cancer: In silico analysis and validation. Exp Mol Pathol. 2021 Dec;123:104688. doi: 10.1016/j.yexmp.2021.104688. Epub 2021 Sep 27. PMID: 34592197.
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Critical roles of microRNA-196 in normal physiology and non-malignant diseases: Diagnostic and therapeutic implications.

Experimental and molecular pathology

Bastami M, Masotti A, Saadatian Z, Daraei A, Farjam M, Ghanbariasad A, Vahed SZ, Eyvazi S, Mansoori Y, Nariman-Saleh-Fam Z.
PMID: 34166682
Exp Mol Pathol. 2021 Oct;122:104664. doi: 10.1016/j.yexmp.2021.104664. Epub 2021 Jun 21.

MicroRNAs (miRNAs) have emerged as a critical component of regulatory networks that modulate and fine-tune gene expression in a post-transcriptional manner. The microRNA-196 family is encoded by three loci in the human genome, namely hsa-mir-196a-1, hsa-mir-196a-2, and hsa-mir-196b. Increasing...

Cite
Bastami M, Masotti A, Saadatian Z, et al. Critical roles of microRNA-196 in normal physiology and non-malignant diseases: Diagnostic and therapeutic implications. Exp Mol Pathol. 2021;122:104664doi: 10.1016/j.yexmp.2021.104664.
Bastami, M., Masotti, A., Saadatian, Z., Daraei, A., Farjam, M., Ghanbariasad, A., Vahed, S. Z., Eyvazi, S., Mansoori, Y., & Nariman-Saleh-Fam, Z. (2021). Critical roles of microRNA-196 in normal physiology and non-malignant diseases: Diagnostic and therapeutic implications. Experimental and molecular pathology, 122104664. https://doi.org/10.1016/j.yexmp.2021.104664
Bastami, Milad, et al. "Critical roles of microRNA-196 in normal physiology and non-malignant diseases: Diagnostic and therapeutic implications." Experimental and molecular pathology vol. 122 (2021): 104664. doi: https://doi.org/10.1016/j.yexmp.2021.104664
Bastami M, Masotti A, Saadatian Z, Daraei A, Farjam M, Ghanbariasad A, Vahed SZ, Eyvazi S, Mansoori Y, Nariman-Saleh-Fam Z. Critical roles of microRNA-196 in normal physiology and non-malignant diseases: Diagnostic and therapeutic implications. Exp Mol Pathol. 2021 Oct;122:104664. doi: 10.1016/j.yexmp.2021.104664. Epub 2021 Jun 21. PMID: 34166682.
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